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Exciting research out of Leipzig University just turned up a promising molecular “switch” for bone strength. The receptor GPR133, when activated by a new compound called AP503, not only halted bone loss in mice—but actually reversed it, even mimicking what mechanical exercise normally does for healthy bones.Current osteoporosis treatments can be messy—with side effects or limited lasting results. This new approach targets the body’s natural bone-building machinery and could one day offer a safer, more regenerative way to age resiliently.
